2C-B (4-Bromo-2,5-dimethoxyphenethylamine)

Psychedelicoral · inferred

Synthetic phenethylamine psychedelic created by Alexander Shulgin. 5-HT2A/2C agonist with effects lasting 4-6 hours. Often described as a gentler psychedelic with more manageable headspace than tryptamines. Metabolized by MAO-A and MAO-B. Shorter duration and half-life than classical psychedelics. Popular at low doses (5-15 mg) for empathogenic effects.

Projected serum levels — 20 mg, as needed (shown daily)

2C-B (4-Bromo-2,5-dimethoxyphenethylamine)
2C-B (4-Bromo-2,5-dimethoxyphenethylamine) modeled serum levels, 20 mg as needed (shown daily) over 15 days Population-based pharmacokinetic estimate. Steady state reached after approximately 1 days. 0 2.5 5 7.5 10 Day 0 Day 4 Day 8 Day 11 Day 15 Time on a regular schedule ≈ steady state · day 1

Maintenance schedule: 20 mg as needed (shown daily) (oral).

Modeled steady state after ~1 days: peak ≈ 5.5 mg, trough ≈ 0.001 mg body load. Population-based estimate over 15 days for a 20 mg dose at a 70 kg reference body mass — the interactive app scales curves to your doses, timing, and body mass.

Key facts

Category
Psychedelic
Route modeled
Oral
Model confidence
inferred
Half-life
2.5 h
Common dose
20 mg
Reference dose range
10–40 mg (single dose)
Suggested cadence
as needed (shown daily)

Documented interactions

  • danger
    1P-LSD (1-Propionyl-LSD) + 2C-B (4-Bromo-2,5-dimethoxyphenethylamine) Serotonin risk

    1P-LSD (1-Propionyl-LSD) and 2C-B (4-Bromo-2,5-dimethoxyphenethylamine) both have serotonergic activity. Risk of serotonin syndrome (hyperthermia, rigidity, autonomic instability, altered mental…

  • danger
    25I-NBOMe + 2C-B (4-Bromo-2,5-dimethoxyphenethylamine) Serotonin risk

    25I-NBOMe and 2C-B (4-Bromo-2,5-dimethoxyphenethylamine) both have serotonergic activity. Risk of serotonin syndrome (hyperthermia, rigidity, autonomic instability, altered mental status).

  • danger
    2C-I (2,5-Dimethoxy-4-iodophenethylamine) + 2C-B (4-Bromo-2,5-dimethoxyphenethylamine) CYP inhibition

    2C-B (4-Bromo-2,5-dimethoxyphenethylamine) reversible_inhibitor of MAO-A predicted to change 2C-I (2,5-Dimethoxy-4-iodophenethylamine) AUC by ~2.00x

  • danger
    3-MeO-PCP (3-Methoxyphencyclidine) + 2C-B (4-Bromo-2,5-dimethoxyphenethylamine) Serotonin risk

    2C-B (4-Bromo-2,5-dimethoxyphenethylamine) and 3-MeO-PCP (3-Methoxyphencyclidine) both have serotonergic activity. Risk of serotonin syndrome (hyperthermia, rigidity, autonomic instability, altered…

  • danger
    4-AcO-DMT (Psilacetin) + 2C-B (4-Bromo-2,5-dimethoxyphenethylamine) Stacked effects

    2C-B (4-Bromo-2,5-dimethoxyphenethylamine) and 4-AcO-DMT (Psilacetin) both push the serotonin 5HT1A, serotonin 5HT2A, and serotonin 5HT2C in the same direction.

  • danger
    5-HTP (5-Hydroxytryptophan) + 2C-B (4-Bromo-2,5-dimethoxyphenethylamine) Serotonin risk

    2C-B (4-Bromo-2,5-dimethoxyphenethylamine) and 5-HTP (5-Hydroxytryptophan) both have serotonergic activity. Risk of serotonin syndrome (hyperthermia, rigidity, autonomic instability, altered mental…

Serum checks 178 modeled interaction pairings for 2c-b (4-bromo-2,5-dimethoxyphenethylamine) across your whole stack — absorption conflicts, enzyme inhibition, and nutrient depletion included.

Research behind this entry

  1. Assessment of the Acute Effects of 2C-B vs. Psilocybin on Subjective Experience, Mood, and Cognition Mallaroni P et al. · Clinical Pharmacology & Therapeutics, 2023 DOI

    Controlled comparison of 2C-B 20mg vs psilocybin in humans showing shorter duration of 2C-B effects (resolving within 6h) with distinct subjective profile and shorter plasma half-life.

  2. Acute Pharmacological Effects of 2C-B in Humans: An Observational Study Gonzalez D et al. · Frontiers in Pharmacology, 2018 DOI

    First systematic documentation of 2C-B acute effects in naturalistic human use, characterizing onset, peak effects, duration, and physiological parameters.

2 published studies referenced in the app, each with a plain-language summary.