Valproic Acid / Divalproex (Depakote)

Anticonvulsantprescriptionoral · inferred

Branched-chain fatty acid anticonvulsant and mood stabilizer with multiple mechanisms: increases GABA via inhibition of GABA transaminase, blocks voltage-gated sodium channels, and inhibits histone deacetylase. First-line for generalized epilepsy, bipolar mania, and migraine prophylaxis. Highly protein-bound (90-95%) with nonlinear kinetics at saturation. Narrow therapeutic index requiring serum monitoring (50-125 mcg/mL). Teratogenic (Category X), neural tube defects. Hepatotoxicity risk especially in children <2 years.

Projected serum levels — 500 mg, twice daily

Valproic Acid / Divalproex (Depakote)
Valproic Acid / Divalproex (Depakote) modeled serum levels, 500 mg twice daily over 15 days Population-based pharmacokinetic estimate. Steady state reached after approximately 2 days. 0 250 500 750 1k Day 0 Day 4 Day 7 Day 11 Day 15 Time on a regular schedule ≈ steady state · day 2
Valproic Acid / Divalproex (Depakote) modeled serum levels with a loading dose of 1.1k mg, then 500 mg twice daily The first dose is larger so levels approach steady state faster. 0 250 500 750 1k Day 0 Day 4 Day 7 Day 11 Day 15 Time on a regular schedule

Maintenance schedule: 500 mg twice daily (oral).

Loading schedule: 1.1k mg on day 1, then 500 mg twice daily — reaching therapeutic levels sooner.

Modeled steady state after ~2 days: peak ≈ 965 mg, trough ≈ 569 mg body load. Population-based estimate over 15 days for a 500 mg dose at a 70 kg reference body mass — the interactive app scales curves to your doses, timing, and body mass.

Key facts

Category
Anticonvulsant
Route modeled
Oral
Model confidence
inferred
Half-life
13 h
Common dose
500 mg
Suggested maximum
3k mg/day
Reference dose range
250–3k mg (single dose)
Suggested cadence
twice daily
Validated against
500 mg oral — Cmax 70 mg/L at 2 h

Documented interactions

  • danger
    Candesartan + Valproic Acid / Divalproex (Depakote) CYP inhibition

    Valproic Acid / Divalproex (Depakote) reversible_inhibitor of CYP2C9 predicted to change Candesartan AUC by ~3.33x

  • danger
    Celecoxib (Celebrex) + Valproic Acid / Divalproex (Depakote) CYP inhibition

    Valproic Acid / Divalproex (Depakote) reversible_inhibitor of CYP2C9 predicted to change Celecoxib (Celebrex) AUC by ~2.97x

  • danger
    Cyclosporine (Sandimmune/Neoral) + Valproic Acid / Divalproex (Depakote) Protein binding

    Valproic Acid / Divalproex (Depakote) may displace Cyclosporine (Sandimmune/Neoral) from plasma protein binding sites, transiently raising free drug concentration. This drug has a narrow therapeutic…

  • danger
    Glipizide (Glucotrol) + Valproic Acid / Divalproex (Depakote) CYP inhibition

    Valproic Acid / Divalproex (Depakote) reversible_inhibitor of CYP2C9 predicted to change Glipizide (Glucotrol) AUC by ~2.47x

  • danger
    Levothyroxine (Synthroid) + Valproic Acid / Divalproex (Depakote) Protein binding

    Valproic Acid / Divalproex (Depakote) may displace Levothyroxine (Synthroid) from plasma protein binding sites, transiently raising free drug concentration. This drug has a narrow therapeutic index;…

  • danger
    Meloxicam (Mobic) + Valproic Acid / Divalproex (Depakote) CYP inhibition

    Valproic Acid / Divalproex (Depakote) reversible_inhibitor of CYP2C9 predicted to change Meloxicam (Mobic) AUC by ~2.20x

Serum checks 232 modeled interaction pairings for valproic acid / divalproex (depakote) across your whole stack — absorption conflicts, enzyme inhibition, and nutrient depletion included.

Research behind this entry

  1. Divalproex sodium versus placebo for acute treatment of mania in bipolar disorder (Bowden 1994) Bowden CL et al. · JAMA, 1994 DOI

    Landmark RCT establishing divalproex as effective for acute mania, with 48% response rate versus 25% placebo, leading to FDA approval for bipolar disorder treatment.

  2. Valproic acid for prophylaxis of migraine: a systematic review and meta-analysis Mulleners WM et al. · Cochrane Database of Systematic Reviews, 2013 DOI

    Cochrane review of 10 RCTs confirming valproate reduces migraine frequency by approximately 50% compared to placebo, with efficacy comparable to topiramate and propranolol.

  3. Clinical pharmacokinetics of valproic acid Perucca E · Clinical Pharmacokinetics, 2002 DOI

    Definitive PK review establishing valproate half-life of 9-16 hours (shorter with enzyme inducers), 90-95% protein binding with saturable kinetics, hepatic metabolism, and the importance of free-fraction monitoring.

3 published studies referenced in the app, each with a plain-language summary.