Dihexa

Peptideoral · inferred

Hexapeptide analog of angiotensin IV (N-hexanoic-Tyr-Ile-(6)-aminohexanoic amide) developed for cognitive enhancement. Remarkably long circulating half-life of ~12.7 days (IV) due to serum stability. Crosses the blood-brain barrier. Activates hepatocyte growth factor (HGF)/MET signaling, promoting synaptogenesis. 10 million-fold more potent than BDNF in promoting neurite outgrowth in vitro.

Projected serum levels — 10 mg, once daily

Dihexa
Dihexa modeled serum levels, 10 mg once daily over 97 days Population-based pharmacokinetic estimate. Steady state reached after approximately 31 days. 0 25 50 75 100 Day 0 Day 24 Day 49 Day 73 Day 97 Time on a regular schedule ≈ steady state · day 31
Dihexa modeled serum levels with a loading dose of 30 mg, then 10 mg once daily The first dose is larger so levels approach steady state faster. 0 25 50 75 100 Day 0 Day 24 Day 49 Day 73 Day 97 Time on a regular schedule

Maintenance schedule: 10 mg once daily (oral).

Loading schedule: 30 mg on day 1, then 10 mg once daily — reaching therapeutic levels sooner.

Modeled steady state after ~31 days: peak ≈ 80.4 mg, trough ≈ 80.1 mg body load. Population-based estimate over 97 days for a 10 mg dose at a 70 kg reference body mass — the interactive app scales curves to your doses, timing, and body mass.

Key facts

Category
Peptide
Route modeled
Oral
Model confidence
inferred
Half-life
13 days
Common dose
10 mg
Suggested maximum
20 mg/day
Reference dose range
5–20 mg (single dose)
Suggested cadence
once daily

Research behind this entry

  1. Evaluation of Metabolically Stabilized Angiotensin IV Analogs as Procognitive/Antidementia Agents McCoy AT et al. · Journal of Pharmacology and Experimental Therapeutics, 2013 DOI

    Foundational study characterizing dihexa as a BBB-permeable, serum-stable AngIV analog with half-life of 12.68 days (IV) that reversed scopolamine-induced cognitive deficits at picomolar concentrations.

  2. AngIV-Analog Dihexa Rescues Cognitive Impairment and Recovers Memory in the APP/PS1 Mouse via the PI3K/AKT Signaling Pathway Li J et al. · Molecular Neurobiology, 2021 DOI

    Demonstrated dihexa rescued spatial learning and memory in APP/PS1 Alzheimer model mice via HGF/MET-PI3K/AKT signaling, reducing amyloid plaque burden and neuroinflammation.

2 published studies referenced in the app, each with a plain-language summary.