Clonidine (Catapres)

Antihypertensive / Alpha-2 Agonistprescriptionoral · inferred

Centrally-acting alpha-2 adrenergic agonist that reduces sympathetic outflow from the brainstem, lowering blood pressure, heart rate, and plasma catecholamines. Also used for ADHD, opioid withdrawal, Tourette syndrome, and menopausal flushing. Abrupt discontinuation causes rebound hypertension due to compensatory upregulation of adrenergic tone — must be tapered over 2-4 days. Half-life 12-16h; 50% excreted renally unchanged. Available as oral tablet and transdermal patch (Catapres-TTS).

Projected serum levels — 0.10 mg, twice daily

Clonidine (Catapres)
Clonidine (Catapres) modeled serum levels, 0.10 mg twice daily over 15 days Population-based pharmacokinetic estimate. Steady state reached after approximately 2 days. 0 0.25 0.50 0.75 1 Day 0 Day 4 Day 7 Day 11 Day 15 Time on a regular schedule ≈ steady state · day 2
Clonidine (Catapres) modeled serum levels with a loading dose of 0.23 mg, then 0.10 mg twice daily The first dose is larger so levels approach steady state faster. 0 0.25 0.50 0.75 1 Day 0 Day 4 Day 7 Day 11 Day 15 Time on a regular schedule

Maintenance schedule: 0.10 mg twice daily (oral).

Loading schedule: 0.23 mg on day 1, then 0.10 mg twice daily — reaching therapeutic levels sooner.

Modeled steady state after ~2 days: peak ≈ 0.19 mg, trough ≈ 0.11 mg body load. Population-based estimate over 15 days for a 0.10 mg dose at a 70 kg reference body mass — the interactive app scales curves to your doses, timing, and body mass.

Key facts

Category
Antihypertensive / Alpha-2 Agonist
Route modeled
Oral
Model confidence
inferred
Half-life
14 h
Common dose
0.10 mg
Suggested maximum
0.60 mg/day
Reference dose range
0.050–0.60 mg (single dose)
Suggested cadence
twice daily
Validated against
0.10 mg oral — Cmax 0.000 mg/L at 3 h

Research behind this entry

  1. Clonidine in patients undergoing noncardiac surgery Devereaux PJ et al. (POISE-2 Investigators) · New England Journal of Medicine, 2014 DOI

    Landmark POISE-2 trial (n=10,010) — perioperative clonidine did not reduce death or nonfatal MI after noncardiac surgery but significantly increased hypotension (RR 1.32) and nonfatal cardiac arrest (RR 3.18).…

  2. Clonidine withdrawal: mechanism and frequency of rebound hypertension Geyskes GG, Boer P, Dorhout Mees EJ · British Journal of Clinical Pharmacology, 1979 DOI

    Definitive early study documenting the clonidine withdrawal phenomenon — rebound hypertension with plasma catecholamine surge, tachycardia, and subjective distress upon abrupt discontinuation. Demonstrated that the…

2 published studies referenced in the app, each with a plain-language summary.